Thursday, May 21, 2026

 

Cities change storms, but the impacts depend on the storm itself



New findings could help reduce urban flood risks and improve planning for extreme weather



Texas A&M University





Cities don’t just change the landscape, they change the weather. According to a new study analyzing tens of thousands of rain events in Texas, whether urban areas make rain worse, lighter or simply different depends strongly on the type of storm.

The research, published in Nature, examines more than 40,000 warm‑season storms that passed over or near Dallas–Fort Worth, Austin, San Antonio and Houston between 1995 and 2017. By sorting storms into distinct categories and tracking their three‑dimensional structure using weather radar, scientists found that the four urban areas strengthen some storms while weakening others.

The findings help explain why previous research on urban rainfall has often produced conflicting results — and why blanket statements about cities being “wetter” or “drier” miss the nuance of how storms actually work.

“Different storms are driven by different physical processes,” said Dr. John Nielsen‑Gammon, a Texas A&M University atmospheric scientist and a co-author of the study. “Once you separate storms by type, the patterns became much clearer.”

Looking at storms one by one

Rather than focusing on long‑term rainfall averages, the researchers took an event‑by‑event approach, tracking individual storms as they formed, moved and dissipated. Using detailed radar data, they grouped storms into five common types:

  • single‑cell thunderstorms
  • larger isolated storms
  • cold frontal storms
  • warm frontal storms
  • tropical systems, including hurricanes

Cities boost pop‑up storms

The strongest and most consistent urban effect appeared in small‑scale thunderstorms, the kind that can pop up quickly on hot summer days.

Across all four cities, these local storms occurred 7% to 31% more often over urban areas than over nearby rural land. Radar data also showed that these storms tended to grow taller and more intense over cities, a sign of stronger upward motion in the atmosphere.

The effect was especially noticeable at night, when cities remain warmer than their surroundings.

“Urban areas hold heat after sunset,” said Nielsen‑Gammon, who is also the Texas State Climatologist. “That retained warmth can continue to fuel storms overnight, when similar storms over rural areas are more likely to weaken.”

The researchers found similar urban effects for larger isolated storms, which last longer and cover more area than single‑cell thunderstorms. These storms were also more frequent over cities and showed stronger rainfall signatures, making them a significant contributor to urban flooding risk.

While these storms are usually short‑lived, they can produce intense rainfall over small areas, increasing the risk of flash flooding in cities, where a lot of the rain can't just soak into the ground.

Cold fronts weaken as they cross cities

The study found a different response for cold frontal storms, which are large, organized rain systems driven by temperature differences between air masses.

Cities did not change how often cold fronts occurred, but they did change their strength. When cold frontal storms passed directly over urban areas, their rainfall intensity declined by roughly 16% to 28% compared with rural surroundings.

The researchers suggest that urban heat and roughness (the disruption buildings and other structures create in wind flow) disrupt the lower part of the storm system as it moves across the city.

“Cold front rainfall is driven by sharp temperature and wind differences,” Nielsen‑Gammon said. “As they move into the warmer and more turbulent urban environment, those contrasts can weaken, reducing rainfall intensity.”

Interestingly, the study found hints that cold fronts sometimes intensify slightly just before reaching a city, likely because the warmer urban air can sharpen the temperature contrast ahead of the front.

Warm fronts and tropical storms respond differently

Warm frontal storms showed modest signs of intensification over cities, though the changes were smaller and less certain than for other storm types.

Tropical systems, including hurricanes, did not show consistent changes in how often they occurred or how intense they were over cities. However, the structure of rainfall shifted slightly, with heavy rain forming lower in the atmosphere over urban areas — a detail that could influence flooding behavior on the ground.

“These larger systems are driven mainly by ocean heat and larger-scale wind patterns,” Nielsen‑Gammon said. “Urban effects don’t disappear, but they’re secondary compared to those factors.”

Why this matters for growing cities

The study’s results suggest that storm type matters for how cities experience extreme weather and for how they should prepare.

Urban planners often rely on averaged rainfall statistics to design drainage systems and flood controls. That approach may miss the fact that the storms most enhanced by cities are short‑duration, high‑intensity events, which are particularly dangerous in urban landscapes.

“If you design only for region-wide averages, you can underestimate the kinds of rainfall that actually cause the most damage,” Nielsen‑Gammon said. “Understanding which storms cities amplify helps planners target the real risks.”

The researchers say storm‑specific insights could improve flood resilience, emergency planning and weather forecasting in urban areas.

“Asking whether cities get more or less rain is the wrong question,” Nielsen‑Gammon said. “The right question is which storms are affected, because that’s what determines the risk people actually face on the ground.”

This research was funded by NASA and the U.S. Department of Energy.

By Lesley Henton, Texas A&M University Division of Marketing and Communications

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Healthcare is facing a moral emergency, argue experts



Time to restore kindness and compassion in healthcare to improve patient and staff well-being




BMJ Group





Healthcare has lost its human, moral, and relational foundations and must reconnect with its core values to improve both patient and staff well-being, argue experts in The BMJ today.

Despite unprecedented advances in diagnostic precision, therapeutic capability, and computational power, a deep paradox exists, say authors Don Berwick, Maureen Bisognano and Bob Klaber. Patients increasingly feel processed rather than cared for, staff report moral distress and loss of meaning, and the workforce is haemorrhaging people at an unsustainable rate.

The core problem, they write, is that we have accumulated extraordinary technical power while quietly losing the human, moral, and relational foundations of care on which its effectiveness ultimately depends.

Several powerful forces have helped create this imbalance, they explain. For instance, in some countries the pursuit of profit has choked healthcare's moral purpose, while across the globe modern healthcare has become an industrialised system that processes patients through standardised protocols in ways that risk disregarding the unique texture of individual lives.

This has happened through an imbalanced emphasis on a "rational" lexicon (focused on measurement, targets and efficiency) over a "relational" one (concerned with feelings, kindness and human connection).

Yet re-establishing the relational balance is not a sentimental or "soft" approach; it is vital for quality and safety, they argue.

They point to research on NHS culture and behaviour that found organisations where staff felt supported and valued had consistently lower patient death rates, while the Institute for Healthcare Improvement (IHI) framework shows that the conditions for increasing joy in work - clarity of purpose, psychological safety, and feeling that what matters to you is actually valued - are both achievable and measurable.

Kindness - linked empirically to better staff retention, higher teamworking scores, and improved patient outcomes - should also be repositioned at the business end of delivering high quality care, they add.

The "What matters to you?" movement, inspired by an article in the New England Journal of Medicine, exemplifies this shift, changing the clinical encounter from a diagnostic focus to a partnership based on the patient's lived reality.

While the forces pulling healthcare away from its human dimension are structural and powerful, they are not irreversible, they say. Every ward round, clinical consultation, and leadership conversation is a small but powerful opportunity for all of us working in healthcare to balance relational practice with rational systems and processes.

The evidence is clear: patients do better and staff thrive when healthcare systems invest in joy, kindness, and compassionate leadership, they write. “We do not need to wait for system reform. We can begin now on our collective leadership challenge to reconnect healthcare with its mission and purpose.”

 

We can help the body fight entire viral families


LJI scientists discover how we can harness T cells to combat deadly infections, including Lassa fever and hantavirus disease




La Jolla Institute for Immunology





LA JOLLA, CA—Scientists at La Jolla Institute for Immunology (LJI) have discovered that combining key vaccine ingredients could give the body the tools it needs to fight the entire family of arenaviruses with a single vaccine. This “pan-arenavirus” vaccine approach may protect against life-threatening infections from Lassa virus, Junin virus, and many other arenaviruses with pandemic potential.

"We are now moving toward the next steps of designing vaccines and seeing which formulations may work best," says LJI Professor Alessandro Sette, Dr.Bio.Sci.

This research comes as scientists are working to stop the spread of Andes hantavirus, which recently caused a deadly outbreak among cruise ship passengers and crewmembers. Hantaviruses and arenaviruses are very similar in how they spread (between rodents and humans) and how they work (both are single-strand, segmented RNA viruses). 

In their Cell Reports Medicine paper, the researchers show that T cells can spot the family resemblance between different species of arenaviruses. Harnessing these "cross-reactive" T cells is critical for designing "pan-viral" vaccines that combat many viruses at once.

"We can apply this new research approach to multiple viral families, no matter how rare they are in the human population," says LJI Research Assistant Professor Alba Grifoni, Ph.D., who co-led the study, with funding from the National Institutes of Health and the Coalition for Epidemic Preparedness Innovations (CEPI).

Investigating a dangerous viral family

Arenaviruses are most common in West Africa countries, where the deadliest arenavirus is called Lassa virus. This virus is spread by a species of rodent, called the multimammate rat. 

Lassa virus kills by causing severe, flu-like symptoms, which can lead to bleeding from the mouth or gastrointestinal tract. The virus kills between 5,000 and 10,000 people each year and hospitalizes many more.

And Lassa virus has several dangerous relatives. The arenavirus family is known for its pandemic potential. Eight arenavirus species are known to cause disease in humans, and there are dozens of other arenaviruses that spread in animal populations.

About 45,000 years ago, arenavirus evolution diverged, leading to marked differences between "Old World" arenaviruses (in European, African, and Asian countries) and "New World" arenaviruses (in the Americas).

For the new study, LJI scientists examined whether T cells could "cross-react" between Old World and New World arenavirus species. After thousands of years, could T cells still spot the family resemblance?

Answering this question is key to pandemic preparedness. 

"Animals carry many viruses, and only some of these viruses will acquire the ability to infect humans and cause an outbreak," says Grifoni. "It's hard to predict which emerging virus might cause disease, which is why it is important to study T cells. "So why do we want to look at T cells? T cells are good at recognizing viruses, even if a virus is mutating."

Taking on Old World arenaviruses

First came an encouraging discovery—the scientists found that human T cells have the ability to cross-react to epitopes shared by all Old World arenavirus species. Epitopes are sites on a virus that T cells can recognize as a sign of infection. Sette and Grifoni found that Old World arenaviruses have shared, or "conserved," epitopes.

That means a vaccine against Lassa virus could potentially also protect against the Old World Lujo virus and lymphocytic choriomeningitis virus (LCMV) for example. In fact, there are already experimental Lassa virus vaccines under development that can induce T cells able to target these shared epitopes that are similar within the Old world viral family.

T cells that targeted Old World arenaviruses did not cross-react to New World arenaviruses. It appears the two groups of viruses are too different and don't share the same epitopes.

New World, new epitopes

It was time to learn more about New World arenavirus epitopes. The scientists began by analyzing T cell responses to Junin virus, an arenavirus that causes Argentine hemorrhagic fever. Some people in Argentina have received a vaccine against Junin virus, which means their bodies have already produced T cells to fight infection. 

In work spearheaded by LJI Postdoctoral Associate YeiI Lee, Ph.D., the scientists discovered that certain T cells that target Junin can cross-react to other New World arenaviruses.

This discovery gives LJI researchers a potential path for developing a broad, "pan-arenavirus" vaccine. Every vaccine includes an immunogen, an ingredient such as a protein or peptide that sparks an immune response against a vulnerable piece of a pathogen. The new study suggests that a future vaccine should include two immunogens: one from an Old World arenavirus and one from a New World arenavirus.

By combining this virus-fighting weaponry, scientists could develop a vaccine that directs T cells to fight every type of infective arenavirus we know of. This broad T cell activity might even help us fight arenaviruses that emerge from animal reservoirs in the future.

Responding to hantaviruses

Lassa virus and Junin virus aren't part of daily life here in the United States—but hantaviruses are becoming more and more of a local concern. Hantavirus is carried by rodents here in California. According to the California Department of Public Health, risk of infection is low, but people should take steps to avoid rodent dropping and contaminated areas.

Recently, attention has turned to the risk of person-to-person hantavirus transmission. In May 2026, an outbreak of the Andes strain of hantavirus sickened 14 and killed three people on an international cruise ship.

Like arenaviruses, hantaviruses are primarily spread by rodents. The Andes hantavirus, which recently spread on the cruise ship MV Hondius, appears to have the unique ability to spread from human to human. 

Also like arenaviruses, hantaviruses are split into Old World and New World species. Importantly, both arenaviruses and hantaviruses are known as single-strand, segmented RNA viruses, which means they carry similar genetic instructions.

Sette says the new study shows how scientists can analyze T cells to devise life-saving vaccine strategies against arenaviruses, hantaviruses, paramyxoviruses (such as measles), togaviruses (such as Chikungunya), and many other threats.

The Sette and Grifoni labs have received important funding from the National Institutes of Health's National Institute of Allergy and Infectious Diseases (NIAID) and from the Coalition for Epidemic Preparedness (CEPI) to lead research toward pandemic preparedness. Each study brings the LJI team closer to developing rapid response tools to analyze viruses and combat future outbreaks.

"LJI is chipping away and generating data for many different viral families of concern," says Sette. "This research lays the foundation for future studies when one might, God forbid, have to move quickly."

Authors of the study, "Decoding the Human CD4+ T cell Epitope Repertoire For Lassa Fever Virus Reveals Novel Pan-mammarenavirus Vaccine Candidates," included Leila Siddiqui, Alison Tarke, Margaret Kim, Tanner Michaelis, Mariah Macias, Elizabeth Phillips, Simon Mallal, Raphael Trevizani, April Frazier, Richard H Scheuermann, Abril Zuniga, Christian Zmasek, Kevin Spurgers, Gideon Akintunde, Spencer W. Stonier, Talita Bianca Gagliardi, Kelly Warfield, Haoyang Li, Kathryn M. Hastie, Erica Ollmann Saphire, and Gene S Tan.

This study was supported by the National Institute of Allergy and Infectious Diseases and the Intramural Research Program, both of the National Institutes of Health, under awards number 75N93024C00056, U19 AI142790, and R21 AI180853; and by CEPI through the CEPI Immunogen Design for Disease X program.

 

Reusable catheters a safe choice that could save NHS millions, study finds




University of Southampton
IMAGE Reusable catheter patients used 35 per cent fewer antibiotics compared to their single-use only counterparts 

image: 

Reusable catheter patients used 35 per cent fewer antibiotics compared to their single-use only counterparts

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Credit: University of Southampton





Reusable catheters are just as safe for patients as single-use ones and do not increase the risk of urinary tract infections, a new study has found.

Thousands of people use the devices to empty their bladders as much as four times a day on average, and the plastic tubes are used just once before being discarded.

Researchers from the University of Southampton followed hundreds of patients for a year to measure the health impact of reusable intermittent urinary catheters against single-use ones.

They discovered that those who tested reusable catheters used 35 per cent fewer antibiotics compared to their single-use only counterparts.

The landmark study, published in the International Journal of Nursing Studies, was funded by the National Institute for Health and Care Research (NIHR).

Experts say the findings could save the NHS millions and cut down on thousands of tonnes of plastic waste produced each year.

Lead author Professor Mandy Fader, in the school of Health Sciences at the University of Southampton, said: “Until now, there has been a real lack of high-quality evidence to support the reuse of catheters.

“But, with our study, we can say for the first time that it is a safe option to switch to reusable catheters.”

For decades, patients in the UK have been strictly advised by healthcare professionals to use single-use catheters to prevent urinary tract infections (UTIs), leaving no choice for those wanting to use the more green-friendly reusable options.

The Southampton study followed 578 participants across a 12-month period, split into two groups – one of which stuck to single-use tubes.

The other group, however, used both the reusable and single-use catheters, and were given a kit to wash them in soap and water, followed by a soak in chlorine solution.

Scientists found 29 per cent of those in the mixed-use group had at least one UTI during the year, compared to about 34 per cent of those who used a fresh one each time.

Study co-author Margaret Macaulay, also from Southampton, said: “The NHS is full of single-use products, and many healthcare professionals caution against reusable items because of the risk of infection.

“However, our results clearly show that reusable catheters can and should be used by the NHS without increasing infection rates.

“People should have the choice to switch depending on their lifestyle needs – and cut down on the throwaway culture in our health service.”

According to the research team, around 100 million single-use catheters are prescribed by the NHS across the UK every year.

They also found that cost of the single-use tubes to taxpayers has risen from £22million in 1998 to around £200million in 2026.

“A box of 30 single-use catheters can cost about £50,” added Southampton Professor of Continence Cathy Murphy.

“Reusable catheters in our trial cost less than 10p per use and can be reused for a month. If just 25 per cent of patients made the switch, the NHS could save millions.”

Professor Marian Knight, NIHR Programme Director of Programme Grants for Applied Research said: "This study is a powerful example of how NIHR-funded research could lead to real-world impact, improving patient choice, saving the NHS millions, and reducing plastic waste.

“These findings will give the NHS the evidence it needs to consider introducing reusable catheters.

“It shows that thousands of people who use catheters every day could choose reusable catheters without concern about an increased risk of infection compared with single-use options, giving patients access to a more environmentally friendly choice."

ENDS
579 WORDS

 

Common food preservatives linked to high blood pressure and heart disease




European Society of Cardiology
Common food preservatives linked to high blood pressure and heart disease 

image: 

Illustration: Common food preservatives linked to high blood pressure and heart disease

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Credit: Mathilde Touvier

 

  • The study of 112,395 people includes detailed analysis of diet and food ingredients.

  • Researchers found eight preservative food additives linked to high blood pressure or cardiovascular disease.

  • The highest risks were in people who ate the most preservatives.

 

Sophia Antipolis, France – 21 May 2026. Eating foods that contain common preservative food additives may increase the risks of high blood pressure and cardiovascular disease, according to research published in the European Heart Journal [1] today (Thursday).

 

The research was led by Dr Mathilde Touvier, a research director at INSERM (the French National Institute for Health and Medical Research), and Anaïs Hasenböhler, PhD student, both from the Nutritional Epidemiology Research Team at the Université Sorbonne Paris Nord and Université Paris Cité, France.

 

Ms Hasenböhler said: “Food preservatives are used in hundreds of thousands of industrially processed foods. Experimental studies suggest that some preservative food additives may be harmful to cardiovascular health, but we have not had enough evidence on the impact of these ingredients in humans. As far as we know, this is the first study of its kind to investigate the links between a wide range of preservatives and cardiovascular health.”

 

The research is part of a larger study, called NutriNet-Santé, and included 112,395 volunteers from across France. Every six months the volunteers told researchers everything they ate and drank over a period of three days.

 

Researchers carried out detailed analyses of the ingredients of all the food and drink, including any preservatives. They also tracked the volunteers’ health for an average of seven to eight years to see if they develop high blood pressure or any cardiovascular disease.

 

Researcher found that 99.5% of the volunteers had consumed at least one food preservative within the first two years of taking part.

 

Overall, they found that people who ate the largest amounts of ‘non-antioxidant’ preservatives had a 29% higher risk of hypertension, compared to those who ate the least, and a 16% higher risk of cardiovascular disease, including heart attack, stroke and angina. People who ate the most antioxidant preservatives had a 22% higher risk of hypertension. Non-antioxidant preservatives are designed to stop harmful microbes, such as mould and bacteria, from growing, whereas antioxidant preservatives are designed to stop oxidation which means the food will not turn brown or become rancid.

 

Researchers also looked at 17 of the most commonly eaten preservatives and found that eight of these were specifically linked to high blood pressure. These were: potassium sorbate (E202), potassium metabisulphite (E224), sodium nitrite (E250), ascorbic acid (E300), sodium ascorbate (E301), sodium erythorbate (E316), citric acid (E330) and extracts of rosemary (E392). Ascorbic acid (E300) was also specifically linked to cardiovascular disease.

 

Dr Touvier added: “This study has some limitations inherent to its observational design. However, the findings are based on highly detailed data, and we have taken account of other factors that can increase or lower the risk of cardiovascular disease. Experimental research in the literature consistently suggested that preservatives may cause oxidative stress in the body or affect the way the pancreas works.

These results suggest we need a re-evaluation of the risks and benefits of these food additives by the authorities in charge, such as the EFSA in Europe and the FDA in the USA, for better consumer protection. In the meantime, these findings support existing recommendations to favour non-processed and minimally processed foods, and avoid unnecessary additives. Doctors and other healthcare professionals play a key role in explaining these recommendations to the public.”

 

The researchers are now looking at how food additives and ultra-processed foods may affect signs of inflammation, oxidative stress, metabolic profile in the blood and the composition of the gut microbiota. This may help them to understand why additives may increase the risks of disease.