Sunday, July 12, 2020


Wall Street Is Making Millions Off Police Brutality


So-called “police-brutality bonds” are a transfer of wealth from Main Street to Wall Street

By Brooke Sweeney
June 24, 2020,

More than 10,000 protesters across the U.S. have been arrested in the wake of George Floyd’s death at the hands of police. Nearly 20 people have died, and many more have suffered permanent injuries. As those numbers rise, so will the lawsuits — and the expensive settlements.

For example, New York City issued more than $237 million for NYPD payouts on legal settlements and judgements in 2018 alone.

When faced with big legal bills or settlements, cities have lots of ways to come up with cash. Some have dedicated funds, some have insurance policies. Others finance their legal obligations by selling bonds, just as they would to raise money for infrastructure or public parks. And that’s where Wall Street makes bank, with next to no risk.

Big banks compete to underwrite — or act as the middle men — on these “general obligation bonds” because they pay out millions in fees. Then, they sell the bonds to high-net-worth individuals and hedge funds, which collect interest as high as 7%. These so-called “police brutality bonds,” as they're colloquially known, “quite literally allow banks and wealthy investors to profit from police violence,” according to a 2018 report from the American Center on Race and the Economy.

The bonds are also backed by the “full faith and credit” of the issuing municipality, which means the city can do just about anything to service that debt — including raising taxes. Since cities rarely default, the bonds are nearly risk-free for investors. And although taxpayers normally foot the bill for police settlements, the added interest on the bonds can nearly double the costs.

It’s a transfer of wealth from Main Street to Wall Street, and city officials are making it happen with almost no oversight from taxpayers.

“It feels intentional. They’re choosing to take money from us, our schools, our public housing, and give it to Wall Street.”

“It feels intentional. They’re choosing to take money from us, our schools, our public housing, and give it to Wall Street,” said Damon Wiliams, a 27-year-old South Side Chicago resident who was arrested during a protest in Hyde Park earlier this month. Williams, alongside other activists and the Cook County public defender’s office, are now suing the city for denying arrestees access to phone calls and their attorneys.

“Meanwhile, there’s a lawless, militarized force being used to surveil Black people,” Williams said.
Between 2008 and 2017, Chicago sold over $700 million in “police brutality bonds,” more than any other city included in the American Center on Race and the Economy’s report. Over that period, investors reportedly collected $1 billion in interest — and taxpayers spent about twice that much servicing the debt.

And then there are the underwriting fees, or the commissions banks get for selling the bonds on behalf of the city. But the payments aren’t spread out over time like the interest. Banks take their cuts of the proceeds even before the deals are done.

In 2017, Goldman Sachs pocketed $1.8 million as the lead underwriter, or book-runner, on Chicago’s $275 million bond sale. Three years prior, Wells Fargo collected $1.72 million in fees on the city’s $450 million GO bond sale.

“It’s generally easy for GO [general obligation] issuers to find someone willing to underwrite their bonds; the underwriting process is very competitive with many investment banks vying for the business,” said R&C Investment Advisors managing director Roberto Roffo.

With cities across the country facing 20% budget shortfalls due to the pandemic, they’ll also likely need these bonds more than ever. In fact, it’s cities like Chicago — which has a near junk credit rating — that will need access to the credit market the most. And they’ll pay higher yields to do it, which makes a sweet deal for investors.


“Defaults are rare in the muni market and even rarer for general governments, which include GO bonds,” wrote Cooper Howard, Charles Schwab’s director of fixed income and income planning. “Although state and local governments face headwinds, such as a potential slowdown in the economy and rising pension burdens, we think defaults among GOs will continue to be rare.”

Right now, the office of Chicago’s comptroller told VICE News that “no bond borrowing is planned, but all options are on the table.”



Is it shady? Yes. Illegal? No.
In the first four months of 2020, Chicago had already paid out $17 million to victims of police violence, according to the city’s expenditure report. Of that, $10 million will go to Tarance Etheredge, who was paralyzed from the waist down after being shot in the back by Chicago police in 2012.

The settlements may be public information, but if a taxpayer or investor wanted to know whether a bond was used to pay for one, it’s not as easy. Every municipality issues and reports their debt for settlements and judgements differently, and some are more transparent than others.

New York, for example, has reams of publicly available information on NYPD settlements, and the office of the city's comptroller told VICE News that they don't use bonds to pay those off. Los Angeles uses the term “judgment obligation bond,” and lets the public know the money is going to pay off lawsuits.

In Chicago, curious citizens need to look at the official GO bond statement, flip to the “sources and uses of funds” section buried in the prospectus, and they still won’t find any mention of specific lawsuits. Is it shady? Yes. Illegal? No.

Chicago last issued one of these bonds in 2017, for $275 million, $225 million of which was earmarked for settlements and judgements. The interest rate on that bond was over 7%. To put that in perspective, interest rates in the U.S. Treasury market are about 1%, and interest rates on New York City’s municipal bonds are about 3%.

That means an investor who bought $10 million of Chicago’s 2017 general obligation bonds would collect almost $8.5 million in interest over the bond’s lifetime. Chicago’s taxpayers, on the other hand, will be paying off that interest until the bond matures in 2029.

“Nothing about Mayor Lori Lightfoot’s politics makes me optimistic about the future,” said Williams, who’s considering suing the city over his treatment and others at the protest. I am, however, optimistic about the grassroots political effort from below that will put pressure on her.”




Protesters With Plastic Pitchforks Just Showed Up to Billionaires’ Mansions in the Hamptons


If some of the nation’s wealthiest were planning on a quiet, leisurely retreat to their Hamptons estates ahead of the July Fourth weekend, they’re off to a bad start.
July 1, 2020




If some of the nation’s wealthiest residents were planning on a quiet, leisurely retreat to their Hamptons estates ahead of the July Fourth weekend, they’re off to a bad start.

A caravan of 200 protesters — some of them armed with plastic pitchforks — showed up outside the Long Island vacation homes of Michael Bloomberg and other wealthy investors Wednesday to decry the nation's rising income inequality, which has only gotten worse since the onset of the coronavirus pandemic. The activists drove in from New York City and targeted the seaside destination where many rich New Yorkers stayed during the coronavirus crisis, to argue that their plight is being ignored.



“Tax the rich, not the poor!” protesters chanted outside the 22,000-square-foot, $20 million mansion of former NYC mayor and presidential candidate Bloomberg. (He owns several other properties, so it’s unclear whether he was home.)

Since March 18, the combined wealth of all U.S. billionaires has surged by $584 billion, according to the progressive think tank Institute for Policy Studies. Jeff Bezos, the founder of Amazon and the richest person in the world, has gained an extra $43.8 billion, according to the group’s analysis. The U.S. stock market is thriving.



At the same time, millions of low-income Americans are either out of a job — with Black and Latinx Americans being particularly hard-hit — or deemed “essential” enough to risk catching the virus while earning low wages. People can’t pay rent and are facing eviction. Lines at food banks are horrifying long. And some states facing their own economic crises, including New York, are mulling budget cuts to the same public education and social services programs that could help lift people out of poverty once the pandemic abates.



“This is primarily about the fact that we’re living in a time where the 0.1% are not only in control of our economy but also our way of life,” said Alice Nascimento, the director of policy at New York Communities for Change, one of the groups leading the protest. “We’re in the middle of a crisis, and ever since the pandemic started, the richest people in the world have only gotten richer.”

Some protesters brought plastic pitchforks because they’re a symbol of “the working-class rising up,” she said.


Protesters included members of the Shinnecock Indian Nation, New York Communities for Change, the New York Taxi Workers Alliance, and others. They’re hoping that Gov. Andrew Cuomo pushes higher taxes on New York's wealthiest residents to pad the state’s budget and avoid cuts to services that could help the poor during a downturned economy, according to Nascimento.

To make their point heard, they also planned to head to the homes of Stephen Ross, a billionaire real estate developer; Stephen Schwarzman, the CEO of the Blackstone Group, a massive real estate investment firm; and Daniel Loeb, the billionaire behind the hedge fund Third Point.

Cuomo’s office did not immediately respond to a VICE News request for comment, although he’s previously been skeptical of raising taxes on the wealthy. A coalition of state legislators has also called for higher taxes on the richest New Yorkers this month, according to Newsday.
Similar protests, organized by groups including the Alliance of Californians for Community Empowerment, will also take place across California later Wednesday, at the homes of real estate magnates and Elon Musk, the CEO of Tesla.

Editor's note: This story has been updated to clarify that the pitchforks protesters carried were plastic. The headline has also been changed.


JUST IN CASE THE PITCHFORKS MADE THE RICH UNEASY SO THEY WOULD SIC THEIR PRIVATE COPS ON PROTESTERS, PLASTIC ONES WERE USED INSTEAD. TOO BAD THE RIGHT WING PROTESTERS WHO BRING REAL GUNS TO PROTESTS ARE NOT FORCED TO USE PLASTIC ONES

Cover: Screenshot via Twitter/New York Communities for Change
Lebanon’s Economic Collapse Is Causing Even More Suffering Than Coronavirus

“Corona isn't scary. What's scary is that the dollar is fluctuating and a poor person can't afford to buy anything.”



July 10, 2020, 


Lebanon’s economy has been steadily crashing for nearly a year, well before the first cases of COVID-19 arrived.

But a strict lockdown, coupled with an already inflating currency, has drastically accelerated the economic meltdown and left many Lebanese and Syrian refugees barely able to make ends meet.

“Corona isn't scary. What's scary is that the dollar is fluctuating and a poor person can't afford to buy anything,” said Haji Abbas Obied, a resident of Bar Elias, a town in Lebanon’s Bekaa Valley.

Obied is among nearly half of the population that's now living below the poverty line. As food prices soar and businesses shut down, her family struggles to afford groceries and medicine. Some even time their grocery purchases when the currency is down.

“When I came to buy groceries, I was shocked by the prices. Prices have more than doubled. Some prices have tripled, unfortunately,” said Rawda Mazloum, who fled from Syria to Lebanon in 2014.

Aid agencies and community groups have stepped in with food and cash support programs. But they're only a band-aid for a national crisis that has outraged the entire country. Nationwide protests broke out in June after the value of the Lebanese lira hit a record low on the black market.

Many demonstrators blocked roads to bring the country to a standstill and call out their government and banks for years of corrupt practices that have contributed to Lebanon becoming the world’s third most indebted country.

With the lira down 80% of its value since October, Lebanon has begun bailout talks with the International Monetary Fund in May. But disagreement around the scale of financial losses have dragged negotiations.

Cover image: Haji Abbas Obied opens her nearly empty fridge in Bar Elias, Lebanon, saying that it's all she has to feed her family of six. (Michael Downey/VICE News)


https://www.vice.com/en_us/article/z3eybj/lebanons-economic-collapse-is-causing-even-more-suffering-than-coronavirus

Higher rates of pain, bleeding found with Essure birth control device

HOW'S THAT 100% SIDE EFFECTS FREE MALE BIRTH CONTROL PILL COMING ALONG

Higher rates of pain, bleeding found with essure birth control device
(HealthDay)—The permanent birth control device Essure is associated with higher rates of chronic lower abdominal or pelvic pain and abnormal uterine bleeding compared with tubal ligation, according to interim results of a postmarket study ordered by the U.S. Food and Drug Administration.
The researchers compared Essure and tubal ligation among more than 1,100 women across the United States and found that rates of chronic lower abdominal or  were just over 9 percent in the Essure group and 4.5 percent in the tubal ligation group, and rates of abnormal uterine bleeding were 16.3 percent in the Essure group and 10.2 percent in the tubal ligation group, CNN reported.
The Essure group had higher rates of gynecologic operations—including surgery to remove the device—than the tubal ligation group, while pregnancy rates were similar in the two groups, according to a statement from Terri Cornelison, M.D., director of the FDA Health of Women Program.
In 2018, Essure maker Bayer pulled the device from the U.S. market due to concerns about side effects. The FDA told Bayer to extend a postmarket surveillance study on Essure from three to five years, CNN reported. The study is ongoing, and patients are still completing one-year follow-up visits, the FDA said.
"As the FDA itself notes: 'This study is ongoing and the results are interim. Final analyses of end points will not be completed until the study concludes' in 2025. It is therefore too early to draw any conclusions," Bayer said in a statement, CNN reported. "The results of several large, real-world observational studies comparing patients with Essure to patients who have had tubal ligations consistently show that Essure's safety profile is similar to that of tubal ligation."
FDA announces safety monitoring measures for the essure device


Global COVID-19 registry finds strokes associated with COVID-19 are more severe, have higher mortality
by American Heart Association
Credit: Unsplash/CC0 Public Domain

Acute ischemic strokes (AIS) associated with COVID-19 are more severe, lead to worse functional outcomes and are associated with higher mortality , according to new research published yesterday in Stroke, a journal of the American Stroke Association, a division of the American Heart Association.

In "Characteristics and Outcomes in Patients with COVID-19 and Acute Ischemic Stroke: The Global COVID-19 Stroke Registry," researchers analyzed data on patients with COVID-19 and AIS treated at 28 health care centers in 16 countries this year and compared them to patients without COVID-19 from the Acute Stroke Registry and Analysis of Lausanne (ASTRAL) Registry, from 2003 to 2019. Researchers sought to determine the clinical characteristics and outcomes of patients with COVID-19 and AIS.


Between January 27, 2020 to May 19, 2020, there were 174 patients hospitalized with COVID-19 and AIS. Each COVID-19 patient with AIS was matched and compared to a non-COVID-19 AIS patient based on a set of pre-specified factors including age, gender and stroke risk factors (hypertension, diabetes, atrial fibrillation, coronary artery disease, heart failure, cancer, previous stroke, smoking, obesity and dyslipidemia). The final analysis included 330 patients total.

In both patient groups, stroke severity was estimated with the National Institute of Health Stroke Scale (NIHSS), and stroke outcome was assessed by the modified Rankin score (mRS). When AIS patients with COVID-19 were compared to non-COVID-19 patients:
COVID-19 patients had more severe strokes (median NIHSS score of 10 vs. 6, respectively);
COVID-19 patients had higher risk for severe disability following stroke (median mRS score 4 vs. 2, respectively); and COVID-19 patients were more likely to die of AIS.

The researchers noted there are several potential explanations for the relationship between COVID-19-associated strokes and increased stroke severity: "The increased stroke severity at admission in COVID-19-associated stroke patients compared to the non-COVID-19 cohort may explain the worse outcomes. The broad, multi-system complications of COVID-19, including acute respiratory distress syndrome, cardiac arrhythmias, acute cardiac injury, shock, pulmonary embolism, cytokine release syndrome and secondary infection, probably contribute further to the worse outcomes including higher mortality in these patients. ... The association highlights the urgent need for studies aiming to uncover the underlying mechanisms and is relevant for prehospital stroke awareness and in-hospital acute stroke pathways during the current and future pandemics."


Explore furtherFollow the latest news on the coronavirus (COVID-19) outbreak

More information: George Ntaios et al, Characteristics and Outcomes in Patients With COVID-19 and Acute Ischemic Stroke, Stroke (2020). DOI: 10.1161/STROKEAHA.120.031208

Journal information: Stroke
Fast-spreading mutation helps common flu subtype escape immune response 
Influenza viruses, like the model shown here, display several kinds of surface proteins on their exteriors. Credit: NIAID

Strains of a common subtype of influenza virus, H3N2, have almost universally acquired a mutation that effectively blocks antibodies from binding to a key viral protein, according to a study from researchers at Johns Hopkins Bloomberg School of Public Health.

The results have implications for flu vaccine design, according to the researchers. Current flu vaccines, which are "seasonal vaccines" designed to protect against recently circulating flu strains, induce antibody responses mostly against a different viral protein called hemagglutinin.


The new mutation, described in the study published online June 29 in PLOS Pathogens, was first detected in the 2014-2015 flu season in some H3N2 flu strains, and evidently is so good at boosting flu's ability to spread that it is now present in virtually all circulating H3N2 strains. Recent flu seasons, in which H3N2 strains have featured prominently, have been relatively severe compared to historical averages.
The mutation alters a viral protein called neuraminidase, and the researchers found in their study that this alteration paradoxically reduces the ability of flu virus to replicate in a type of human nasal cell that it normally infects. However, the researchers also found evidence that the mutation more than compensates for this deficit by setting up a physical barrier that hinders antibodies from binding to neuraminidase.

"These findings tell us that flu vaccines focusing on the hemagglutinin protein are leaving the virus openings to evolve and evade other types of immunity," says study senior author Andrew Pekosz, Ph.D., professor and vice chair of the Department of Molecular Microbiology and Immunology at the Bloomberg School.

Every year, influenza viruses sicken millions of people around the world, killing several hundred thousands. The diversity of flu strains and their ability to mutate rapidly—two strains infecting the same host can even swap genes—have made flu viruses an especially difficult target for vaccine designers. Although scientists are working towards a universal vaccine that will protect long-term against most flu variants, current flu vaccines are designed to protect against only a short list of recently circulating strains. Any mutation that occurs in these circulating strains and appears to improve their ability to spread is naturally of interest to flu virologists.

The goal of the study was to understand better the workings of the new H3N2 mutation. Scientists have known that it alters the flu virus's neuraminidase protein in a way that provides an attachment point, close to neuraminidase's active site, for a sugar-like molecule called a glycan. But how the presence of a glycan at that location on the neuraminidase protein improves the virus's ability to infect hosts and spread hasn't been clear.
Pekosz and first author Harrison Powell, Ph.D., a graduate student in his laboratory at the time of the study, compared the growth, in laboratory cells, of typical H3N2 strains that have the glycan-attachment mutation to the growth of the same flu strains without the mutation. They found that the mutant versions grew markedly more slowly in human cells from the lining of the nasal passages—a cell type that a flu virus would initially infect.

The researchers found the likely reason for this slower growth: the glycan-attracting mutation hinders the activity of neuraminidase. The protein is known to serve as a crucial flu enzyme whose functions include clearing a path for the virus through airway mucus, and enhancing the release of new virus particles from infected cells.

It wasn't entirely unexpected that the addition of a moderately bulky glycan molecule near the enzyme's active site would have this effect. But it left unexplained how that would benefit the virus.

The scientists solved the mystery by showing that the glycan blocks antibodies that would otherwise bind to or near the active site of the neuraminidase enzyme.

Neuraminidase, especially its active site, is considered one of the most important targets for the immune response to a flu infection. It is also the target of flu drugs such as Tamiflu (oseltamivir). Thus it makes sense that a mutation protecting that target confers a net benefit to the virus, even if it means that the neuraminidase enzyme itself works less efficiently.

The finding highlights the potential for flu viruses to evade therapies, seasonal vaccines, and the ordinary immune response, Pekosz says, and points to the need for targeting multiple sites on the virus to reduce the chance that single mutations can confer such resistance.

The researchers have been following up their findings with studies of how the new mutation affects the severity of flu, how it has spread so rapidly among H3N2 strains, and how these altered flu strains have adapted with further mutations.

"Neuraminidase antigenic drift of influenza A virus H3N2 clade 3c.2a viruses alters virus replication, enzymatic activity and inhibitory antibody binding" was written by Harrison Powell and Andrew Pekosz.


Explore furtherHow the flu vaccine fails
More information: Harrison Powell et al. Neuraminidase antigenic drift of H3N2 clade 3c.2a viruses alters virus replication, enzymatic activity and inhibitory antibody binding, PLOS Pathogens (2020). DOI: 10.1371/journal.ppat.1008411
Journal information: PLoS Pathogens
WHAT WE DON'T KNOW ABOUT COVID-19 
Long tail of coronavirus can prolong suffering for months
JULY 10, 2020
Credit: CC0 Public Domain

They call themselves "long-haulers", "long-tailers", or simply survivors.

Some have been sick almost as long as the new coronavirus is known to have existed.

Six months after the virus began to scorch its way across the planet, it is becoming clear that COVID-19 causes far more symptoms than first suspected.

Thousands of people of all ages are staying sick for weeks or even months.
British forensic psychiatrist Jenny Judge began an odyssey of illness in March with a fever, cough, headache and breathing problems.

She has since experienced waves of other symptoms including a racing heart, scalding rashes and "COVID toes", which were itchy and ulcerated.

At one point she was so delirious she heard her dogs talking, and was not particularly surprised.

"Now I am going through a belly phase," she told AFP on day 111 of her ordeal.

More than 12 million COVID-19 cases have been recorded worldwide with more than 550,000 deaths. Some six million people are listed as "recovered".

But these figures do not tell the full story.

'They feel left out'

A study of 143 recovered hospital patients in Italy, published in the JAMA Network journal on Thursday, found that 87 percent were still suffering at least one symptom 60 days after falling ill.

Fatigue and breathing difficulties were most common.

This follows research published last week by the US Centers for Disease Control and Prevention that found of 350 people surveyed, about 60 percent of inpatients and around a third of outpatients were not back to health 14-
21 days after testing positive.

People leaving hospital may need ongoing care for organ damage, injuries sustained in invasive oxygen therapy or post-traumatic stress.

But those who have coped with their illness at home often do not have an explanation for their continuing symptoms, and may face scepticism or outright disbelief from employers and doctors.

"I think these people feel very left out and that nobody's looking after them," said Tim Spector, Professor of Genetic Epidemiology at King's College London, who is behind a large-scale symptoms-tracking project.

"Some of them can have really debilitating fatigue."
Some 3.8 million people in the UK have logged on to the app since it was launched in March, while it also has more than 300,000 users in the US and 186,000 in Sweden.

Researchers think that up to one in 10 of them still have symptoms after 30 days and some remain unwell for months.

Spector, who estimates there may be a quarter of a million people in the UK with longer-term illness, receives around 10 emails a day from people who are still ill and who feel "no one is listening to them".

Part of the problem is the sheer variety of symptoms, many of which do not appear in official health advice.


"I used to be a rheumatologist and study very rare autoimmune diseases like lupus which can affect any part of the body and can present in different ways—but this is even more weird," he said, adding the app has identified 19 symptoms so far.

"You can have people just with skin problems. You can get people probably just with diarrhoea and chest pain. It's really very unusual."

'Might be you'
COVID support groups are attracting thousands of members on social media and hashtags are trending in languages including Japanese, French, English and German.

Many people posting in these groups say they have experienced disbelief from doctors or employers.

Those who became ill in March may face particular problems as testing was scarce and they may have no clear evidence that they were ever infected.

Judge said even though she is a doctor she has faced scepticism from staff at her local hospital, with one medic suggesting her high heart rate might be anxiety.

She believes this is partly because hospital doctors are only just coming into contact with patients whose initial symptoms were not considered serious enough for emergency treatment.

But the 48-year-old, who had no pre-existing conditions, said there could also be an element of denial at play.

"If you accept the person who looks just like you, who is a doctor, who was taking all the precautions, is sick at a hundred plus days down the line—that might be you," she said.

'Sick and struggling'


The situation is improving with new studies launched and a growing number of people sharing their stories.

Paul Garner, a professor of infectious diseases at Liverpool School of Tropical Medicine, began a blog in the British Medical Journal out of frustration.

He had been ill for a month but the health advice he had read online said the illness lasted just two weeks.

The previously fit and healthy 64-year-old was tormented with blinding headaches, shortness of breath and a strange tingling in his arms and legs that he said is like the "fizziness" of Sichuan peppercorns.

At one point he thought he was losing consciousness: "I thought I was dying, that was how scary it was."

Garner said one of the hardest things about his illness has been the "muddling" in his head and mood swings.

"This doesn't happen to me, I don't get depressed," he said, adding he had sought advice from a rehabilitation consultant, who said depression was a possible side effect.

"I was just in tears, but it kind of helped me understand what was going on."

He endured several false dawns. On day 45, after he had felt better for a few days, Garner decided he had finally shaken off the virus and tested the waters with a workout in his front room.

"Then bang! Monday: 'felt rotten all day, consequence of exercise'," he said, reading from his diary.

"
It knocked me back a week."

With the help of literature for ME (myalgic encephalomyelitis) and chronic fatigue, he devised a routine interspersing light physical or mental exertion with periods of rest.

Speaking to AFP on day 96 of his illness, Garner said he was gradually seeing improvement.

But he is concerned that vulnerable people may be pressured into trying to return to work before they are ready.

"Everybody's obsessed about the public health control. But what about the people that are sick and struggling and not knowing what's going on?" he said.

Risking it?

It is not yet clear whether long-lasting symptoms are caused by the virus itself or the body's overzealous immune reaction.

Spector said some of the long-haulers may still have traces of virus in their systems, although it is unclear whether they could still be infectious.

"There will soon be these rapid tests in airports, does that mean they will never be able to travel, because they'll be positive all the time?" he said.

Other diseases can cause prolonged "post-viral" effects.

A 2009 study of 233 people who had been treated in hospital for SARS, another coronavirus, found that four years after their illness 40 percent reported suffering from depression or chronic fatigue.

"The implication for rehabilitation and appropriate support for the SARS/COVID-19 victims is obvious," said Yun Kwok Wing, a professor at the Chinese University of Hong Kong who was one of the authors of that study.

As we learn more about the new coronavirus, our perception of the risks of the disease may need to stretch beyond the likelihood of dying.

Young people are still most likely to get a mild version of the disease, but Judge said they should also be aware that if they do catch COVID-19 there is a chance they could be ill for months.

"It seems to be a kind of Russian roulette type thing, we don't yet know what's making some people get a longer illness," she said.

"There's a lot to learn still."